Scaling Limit of a Discrete Prion Dynamics Model
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چکیده
The modelling of intracellular prion infection has been dramatically improved in the past few years according to recent progress in molecular biology of this pathology. Relevant models have been designed to investigate the conversion of the normal monomeric form of the protein (denoted PrPc) into the infectious polymeric form (denoted PrPsc) according to the auto-catalytic process: PrPc + PrPsc −→ 2PrPsc,
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تاریخ انتشار 2009